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In general, the data obtained are consistent with the conclusions made earlier by other authors [1, 2] regarding the involvement of oxidative stress in endometriosis progression

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This report describes opposing effects of synaptic compared with extrasynaptic NMDARs on BDNF expression and cell survival: the activation of synaptic NMDARs led to upregulation of BDNF expression and the activation of extra-synaptic NMDARs reduced BDNF expression

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