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The L-cells in the intestinal epithelium express various nutrient-sensing receptors: Dietary fats are particularly potent GLP-1 secretagogues, activating free fatty acid receptors (such as GPR40 and GPR120) on L-cells Proteins and amino acids stimulate GLP-1 through peptide-sensing mechanisms and calcium-sensing receptors Carbohydrates trigger release via glucose transporters and sweet taste receptors expressed on enteroendocrine cells Dietary fibre , particularly soluble fibre, promotes GLP-1 secretion through fermentation products (short-chain fatty acids) produced by gut microbiota The rate and site of nutrient delivery also influences GLP-1 response

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A probable-category attribution is what makes the case clinically useful, and the rest of this discussion follows from it

It is less accurate than DXA but is far better than relying on scale weight alone