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Moreover, Wnt signaling is responsible for the development of acquired anti-c-Met therapy resistance since its components are upregulated in resistant cells [107]

This article provides an overview of pathophysiological mechanisms in tendon disease

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In contrast, H3R2 modification (H3R2me1/me2s) by PRMT5 is associated with transcriptional activation [57,58], which activates and recruits WD-40 repeat-containing protein 5 (WDR5) to promote H3K4me3 and gene transcription [59,60]