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Special population considerations: Metabolic syndrome and prediabetes Those with metabolic dysfunction represent primary candidates for MOTS-c based on its core mechanisms

Ex-4c stimulates GLP-1Rs to directly activate arcuate POMC neurons While POMC neurons are present in both the nucleus tractus solitarius (NTS) and the ARH, previous studies have shown that NTS POMC neurons do not express GLP-1Rs [15]

Discussion In present study, we find that dysfunction of mitophagy may play an important role in pressure overload-induced cardiac hypertrophy, and semaglutide ameliorates this type of cardiac hypertrophy by improving mitophagy to suppress the activation of NLRP3 inflammasome