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doi: 10.3390/biomedicines12051001
All these models have provided good insights into iodine deficiency in goiter development and thyroid dysfunction and are critical to an understanding of the global public health problem of iodine deficiency-related thyroid disorders (Kujur et al
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From an immunological perspective, these associations arise from a breakdown in self-tolerance mediated by overlapping T-cell and B-cell autoreactivity, shared cytokine pathways (e.g., IFN-, IL-18), and common antigen-presenting HLA alleles
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