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In addition, GSSG retained in mitochondria during oxidative stress can lead to the S-gluthionylation of target proteins with mitochondrial dysfunction (20, 21)

For this, nodule structure and gene expression profilesincluding the expression of genes associated with nodule development ( EFD , NF-YA1 ), (h)GSH biosynthesis ( GSH1 , GSHS , and hGSHS ), markers of defense responses ( PR1 , PR10 ), and nodule senescence markers ( TPP , Cyp15a )were compared among untreated, BSO-treated, and BSO+GSH-treated roots with nodules (Figure 7)

see Methods for details)

Almazan et al., [83] indicated that the intrahippocampal administration of a TNFR2-specific blocking antibody improved recognition memory, spatial memory, and synaptic potentiation in response to chemically induced LTP (cLTP) in 5xFAD mice